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During "test systems" studies, GLP is also necessary in regards to handling live organisms or microorganisms, as well as plants. Taken together, these studies suggest that, with similar energy contents, diets that are richer in MUFAs or omega-3 polyunsaturated fatty acids (PUFAs) than in SFAs, could increase GLP-1 secretion from L-cells, which may be a mediator for the increase in insulin secretion, insulin sensitivity, β-cell proliferation, as well as the improved glucose tolerance observed in animal models and in humans. The 2016 research review previously mentioned suggests that specific nuts may increase GLP-1 levels through their protein, fiber, and healthy fat content. At low doses the peptide suppresses fat intake while at high doses it exhibits either no effect or in some experiments high doses stimulate fat intake. GLP-1 and glucose insulinotropic peptide blood concentrations following the addition of 85.0 g of pistachios to a carbohydrate-containing meal in adults with metabolic syndrome. GLP-1 blood concentrations following the addition of 42.5 g of almonds to a carbohydrate-containing meal, which could be due to the small sample size of the study. GLP-1 blood concentrations with the addition of 42.5 g of peanuts or peanut butter to a carbohydrate-containing meal, it is also possible that higher quantities of nuts are necessary to induce a sufficient rise in GI hormone secretion.
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